From 1a51da139633fe24597eb9b74056bae9b87f8a6f Mon Sep 17 00:00:00 2001 From: Charles Plessy Date: Fri, 18 Mar 2011 13:31:56 +0900 Subject: [PATCH] =?utf8?q?=20#=20Un=20papier,=20un=20caf=C3=A9.?= MIME-Version: 1.0 Content-Type: text/plain; charset=utf8 Content-Transfer-Encoding: 8bit --- biblio/21297615.mdwn | 3 +++ biblio/21382548.mdwn | 3 +++ biblio/21382552.mdwn | 3 +++ 3 files changed, 9 insertions(+) create mode 100644 biblio/21297615.mdwn create mode 100644 biblio/21382548.mdwn create mode 100644 biblio/21382552.mdwn diff --git a/biblio/21297615.mdwn b/biblio/21297615.mdwn new file mode 100644 index 00000000..d1dc1a3f --- /dev/null +++ b/biblio/21297615.mdwn @@ -0,0 +1,3 @@ +[[!meta title="DICER1 deficit induces Alu RNA toxicity in age-related macular degeneration."]] +[[!tag not_read repeats miRNA]] +[[!pmid 21297615 desc="Pathology modelled in mouse with the B1 and B2 repeats."]] diff --git a/biblio/21382548.mdwn b/biblio/21382548.mdwn new file mode 100644 index 00000000..c018ec48 --- /dev/null +++ b/biblio/21382548.mdwn @@ -0,0 +1,3 @@ +[[!meta title="Cytoplasmic Intron Sequence-Retaining Transcripts Can Be Dendritically Targeted via ID Element Retrotransposons."]] +[[!tag repeats dendrite]] +[[!pmid 21382548 desc="Proper secondary structure looks necessary."]] diff --git a/biblio/21382552.mdwn b/biblio/21382552.mdwn new file mode 100644 index 00000000..28c08f8b --- /dev/null +++ b/biblio/21382552.mdwn @@ -0,0 +1,3 @@ +[[!meta title="Phosphorylation Regulates OLIG2 Cofactor Choice and the Motor Neuron-Oligodendrocyte Fate Switch."]] +[[!tag neurogenin transcription_factor heterodimer not_read]] +[[!pmid 21382552 desc="“Wild-type OLIG2 forms stable homodimers, whereas mutant (unphosphorylated) OLIG2(S147A) prefers to form heterodimers with Neurogenin 2 or other bHLH partners”"]] -- 2.47.3