From: Charles Plessy Date: Fri, 18 Mar 2011 04:31:56 +0000 (+0900) Subject: # Un papier, un café. X-Git-Url: https://source.charles.plessy.org/?a=commitdiff_plain;h=1a51da139633fe24597eb9b74056bae9b87f8a6f;p=source%2F.git # Un papier, un café. --- diff --git a/biblio/21297615.mdwn b/biblio/21297615.mdwn new file mode 100644 index 00000000..d1dc1a3f --- /dev/null +++ b/biblio/21297615.mdwn @@ -0,0 +1,3 @@ +[[!meta title="DICER1 deficit induces Alu RNA toxicity in age-related macular degeneration."]] +[[!tag not_read repeats miRNA]] +[[!pmid 21297615 desc="Pathology modelled in mouse with the B1 and B2 repeats."]] diff --git a/biblio/21382548.mdwn b/biblio/21382548.mdwn new file mode 100644 index 00000000..c018ec48 --- /dev/null +++ b/biblio/21382548.mdwn @@ -0,0 +1,3 @@ +[[!meta title="Cytoplasmic Intron Sequence-Retaining Transcripts Can Be Dendritically Targeted via ID Element Retrotransposons."]] +[[!tag repeats dendrite]] +[[!pmid 21382548 desc="Proper secondary structure looks necessary."]] diff --git a/biblio/21382552.mdwn b/biblio/21382552.mdwn new file mode 100644 index 00000000..28c08f8b --- /dev/null +++ b/biblio/21382552.mdwn @@ -0,0 +1,3 @@ +[[!meta title="Phosphorylation Regulates OLIG2 Cofactor Choice and the Motor Neuron-Oligodendrocyte Fate Switch."]] +[[!tag neurogenin transcription_factor heterodimer not_read]] +[[!pmid 21382552 desc="“Wild-type OLIG2 forms stable homodimers, whereas mutant (unphosphorylated) OLIG2(S147A) prefers to form heterodimers with Neurogenin 2 or other bHLH partners”"]]